October 1, 2026
The carotid arteries are the primary highways carrying oxygenated blood from the heart through the neck to the brain. A carotid artery dissection occurs when a tear develops in the innermost layer (the intima) of the arterial wall.
Blood under high pressure seeps into the layers of the artery wall, creating a blood clot within the wall itself (intramural hematoma). This can cause the artery to narrow, block blood flow entirely, or form a bulging pseudoaneurysm. Carotid artery dissection is one of the leading causes of ischemic stroke in young and middle-aged adults.
Dissections can develop spontaneously or follow mechanical force to the neck:
Symptoms of carotid dissection often develop in two distinct stages: local warning signs followed by brain ischemia.
If the blood clot reduces cerebral blood flow or breaks free to travel into the brain, it triggers a Transient Ischemic Attack (TIA) or Acute Stroke:
Carotid artery dissection requires rapid evaluation using CT Angiography (CTA) or Magnetic Resonance Angiography (MRA) to visualize the tear and vessel narrowing.
Treatment focuses on preventing stroke. Many dissections heal naturally over time with antiplatelet or anticoagulant medications. However, if a patient experiences persistent blood flow restriction or an expanding pseudoaneurysm, neuroendovascular stenting is performed to expand the vessel lumen, seal the tear, and restore clear blood flow to the brain.